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DALK (Deep Anterior Lamellar Keratoplasty)

Deep Anterior Lamellar Keratoplasty (DALK) is a specialized partial-thickness corneal transplant that replaces diseased outer and middle corneal layers (the epithelium and stroma) while preserving the patient's own healthy inner layers (Descemet's membrane and endothelium). By retaining the host endothelium, DALK virtually eliminates the risk of endothelial graft rejection, provides superior long-term tectonic stability, and significantly reduces intraocular surgical risks compared to full-thickness penetrating keratoplasty. It is the preferred surgical option for advanced keratoconus, corneal ectasia, and stromal dystrophies or scars where the innermost corneal layer remains healthy.

  • Surgery Name

    DALK (Deep Anterior Lamellar Keratoplasty)

  • Speciality

    Ophthalmology (Cornea & Anterior Segment)

  • Duration

    Usually 60–120 minutes

  • Anaesthesia

    Local anaesthesia with sedation, or general anaesthesia

  • Hospital Stay

    Day-care procedure or short overnight stay

  • Recovery Time

    Initial healing in weeks; visual stabilization over 6–12 months

  • Procedure Type

    Partial-thickness (lamellar) corneal transplant

  • Insurance Coverage

    Usually covered when medically indicated

  • Cost Range in India

    Varies based on hospital tier, donor tissue source, and technique

  • Success Rate

    High graft survival (>90% at 5 years); eliminates endothelial rejection

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Symptoms and Signs of Corneal Conditions That May Be Treated with DALK

DALK is indicated for progressive diseases affecting the corneal stroma where endothelial function is preserved. Symptoms arise from structural distortion, irregular astigmatism, or stromal opacification, and may include:

  • Progressive blurring, haziness, or visual distortion (ghosting and monocular polyopia) that cannot be satisfactorily corrected with spectacles.
  • Frequent prescription changes with rapidly escalating irregular corneal astigmatism and myopia.
  • Severe glare, halos around lights, and poor contrast sensitivity, particularly in low-light and night driving conditions.
  • Contact lens intolerance or inability to achieve a stable fit with rigid gas permeable (RGP) or scleral contact lenses due to advancing corneal steepening.
  • Visible corneal opacification, haze, or focal stromal scarring resulting from prior keratitis, trauma, or stromal dystrophies.
  • Sudden vision loss, severe pain, and corneal clouding if acute corneal hydrops occurs due to a break in Descemet's membrane in advanced keratoconus.